coagulation thrombosis
英 [kəʊˌæɡjuˈleɪʃn θrɒmˈbəʊsɪs]
美 [koʊˌæɡjuˈleɪʃn θrɑːmˈboʊsɪs]
网络 凝血性血栓形成
双语例句
- Comparison between domestic bivalirudin and heparin on coagulation and thrombosis in rats
比较国产比伐卢定与肝素对大鼠颈总动脉血栓模型凝血系统的影响 - Effects of Hirudin by Aerosol on Blood Coagulation Function and Thrombosis Formation in Rats
水蛭素气雾给药对大鼠凝血功能和体内血栓形成的影响 - The Observation of the Change of Blood Coagulation Function and the Deep Venous Thrombosis after Abdominal Surgery
腹部手术后凝血功能变化与深静脉血栓形成的观察 - Dynamic Changes of Blood Coagulation System on the Cerebral Cortex Microvascular Thrombosis Induced by Photochemical Method in Rats
光化学法诱导大鼠脑皮质微血管梗塞凝血系统动态变化的研究 - ( 3) The rough surface of ruptured or semi-ruptured plaque activates the plate-lets and coagulation factors to cause thrombosis;
破裂或半破裂的斑块表面粗糙，血小板和凝血因子被激活，形成血栓； - OBJECTIVE: Through observation on its coagulation time in mice and venous thrombosis in rats to study the pharmacodynamics of low molecular weight heparin ( LMWH).
目的：通过观察低分子量肝素（LMWH）对小鼠凝血时间及大鼠静脉血栓形成的影响，对其主要药效学进行了研究，并与国外同类产品Clivarin及普通肝素进行了比较。 - Purpose To investigate the relationship between the tissue factor coagulation pathway and the mechanisms of atrial thrombosis in patients with atrial fibrillation ( AF) by measuring the plasma levels of tissue factor ( TF) and tissue factor pathway inhibitor ( TFPI) antigens.
目的检测心房颤动（AF）患者血浆组织因子（TF）和组织因子途径抑制物（TFPI）抗原的变化，探讨组织因子凝血途径在心房血栓形成机制中的作用。 - Study of Si Miao Yong An Extract on Blood Coagulation and Thrombosis
四妙勇安提取物对血液凝固和血栓形成的影响 - Conclusions The changes of tissue factor coagulation pathway were involving in the mechanisms of atrial thrombosis in patients with atrial fibrillation.
结论心房颤动患者血浆组织因子途径改变表现为凝血活性增高和抗凝活性减低，组织因子凝血途径参与了心房颤动患者心房血栓形成机制。 - Conclusion The increase of plasma levels of D-dimer and Lipoprotein ( a) in patients with ICVD, is related to the enhanced activity of coagulation and high rate of thrombosis.
结论ICVD患者血D二聚体、LP（a）水平升高，这与体内血液凝固性增强和高发血栓形成有关。 - Conclusions Apoptosis and necrosis of VECs and SECs occur in portal area in patients with portal hypertension, which activates the blood coagulation system, promotes thrombosis and destroys hepatic microcirculation.
结论肝硬化门静脉高压症病人门管区内皮细胞和肝窦壁内皮细胞凋亡和坏死明显增加，内皮细胞的凋亡和坏死激活凝血系统，促使血栓形成，从而破坏了肝脏微循环。 - Conclusion: There is strong relationship between the high level homocysteine ( which were caused by lack of the folic acid and vitamin B12) and the change of the coagulation, anticoagulation, fibrinolytic system, and it plays an important role in the thrombosis.
结论：由于叶酸、维生素B12缺乏所致的高同型半胱氨酸血症与脑梗死患者凝血、抗凝血、纤溶系统的异常显著相关，对促进血栓形成具有重要影响。 - Conclusions Persistent high levels of molecular markers on coagulation and fibrinolytic system would be easy to lead to thrombosis in patients with infiltrative metastasis.
结论恶性肿瘤患者术后凝血和纤溶系统指标持续维持在较高的水平，若发生局部浸润、淋巴结或远处脏器转移时，极易发生血栓形成，术后必需加强凝血和纤溶系统指标监测。 - Conclusion Jiuxin capsule has a good effect in protecting endotheliocyte injury by regulating blood coagulation and preventing the thrombosis secondary to the coronary atherosclerosis.
[结论]救心胶囊保护内皮细胞，参与血液凝固性调节，防止冠状动脉粥样硬化继发血栓形成。 - Conclusions There exists abnormally activated hyper-coagulation state in patients with AMI and patients with UAP. TF is recognized as the initial trigger of the extrinsic coagulation pathway which plays a critical role in the pathogenesis of coronary thrombosis.
结论AMI、UAP患者存在异常激活的高凝状态，TF触发的外源性凝血途径，在冠状动脉血栓形成中发挥作用。 - Conclusion There are activation of coagulation and abnormality of anticoagulant system in patients with SLE, and protein C system plays a major role in pathogenesis of the thrombosis in patients with SLE.
结论SLE患者存在凝血的激活和抗凝系统的异常，蛋白C系统在SLE患者的血栓形成中起着重要作用。 - Functions of platelet, coagulation, anticoagulation and fibrinolysis were studied in 47 patients with cerebral thrombosis.
对47例脑血栓形成患者的血小板、凝血、抗凝和纤溶功能的变化进行了研究。 - Coagulation has great influence to thrombosis.
凝血过程在血栓形成中发挥着极为重要的作用。 - Lloyed mixture has anti thrombotic effect, blood coagulation and platelet activation were inhibited, and the ability of vascular endothelial cells against the process of thrombosis was enhanced.
其抗血栓形成机理可能与抗凝、抑制血小板活化和增加血管内皮细胞抗血栓能力有关。 - Severe UC patients had high blood coagulation state. High blood coagulation state and ( or) intestinal micro thrombosis may be one of the pathogenesis of UC.
溃结患者存在血液高凝状态，血液高凝状态和（或）肠黏膜微小血栓形成可能是溃结的发病机制之一。 - Objective: Upon plaque rupture, platelet adhesion, activation and aggregation, and coagulation activation by tissue factor, leading to intracoronary thrombosis, are fundamental mechanisms of acute coronary syndromes ( ACS).
目的：血小板粘附、激活、聚集和组织因子（TF）激活凝血系统、活化凝血酶是伴随斑块破裂冠脉内急性血栓形成进而引发急性冠脉综合征（ACS）的基本机制。 - Results: Disseminated intravascular coagulation was defined by International Society of Thrombosis and Hemostasis criteria ( ISTH). A total of 71 patients ( 44.1%) were diagnosed with DIC according to the gold standard.
结果：根据国际血栓与止血委员会制定的标准（ISTH），共有71例（44.1%）诊断为DIC。 - Blood coagulation is a very complex process that can cause some serious complications, such as resulting in the formation of thrombosis and embolism in the human circulation system.
血液凝固是种极为复杂的过程。不正常的凝血会导致一些严重的并发症，例如增加出血的几率、造成血栓的形成或栓塞于人体循环系统。 - A result of research showed that LPS and IL-1B can increase the expression of tissue factor, which can lead to blood coagulation in vascular endothelial cells and thrombosis. Linoleic acid and linolenic acid can inhibit the expression of the tissue factor.
还有研究认为LPS和IL-1β能增加组织因子的表达，这种组织因子能引发血液在血管内皮细胞表面凝结而形成血栓，亚油酸和亚麻酸能抑制组织因子的表达。